How does an understanding of the guard hypothesis change the way we view host-pathogen dynamics
How does an understanding of the guard hypothesis change the way we view host-pathogen dynamics
So what about evolution?
What happens next?
Most plants are resistant to most pathogens
Non-Host Resistance is likely based on a number of mechanisms
The plant basal defence system
MAMPS are recognized by receptors at the cell surface
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FLS2, the flagellin receptor was identified in Arabidopsis
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So what’s the difference between MAMPs and AVR/Effector genes and between MAMP receptors and R-genes?
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RIN4 again
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Non-host resistance